Rang & Dale’s Pharmacology 7th Edition by Humphrey P. Rang – Test Bank
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Rang & Dale’s Pharmacology 7th Edition by Humphrey P. Rang – Test Bank
Rang: Rang & Dale’s Pharmacology, 7th Edition
Test Bank
Chapter 5: Cell proliferation, apoptosis, repair and regeneration
MULTIPLE CHOICE
- 1. In the death receptor pathway, activation of tumor necrosis factor receptors causes the receptors to trimerize and complex an adapter protein, thereby leading to activation of
- a. mitochondrial pathways
- b. caspase-8
- c. pro-apoptotic factor Bcl-2
- d. a p53 protein
- e. DNA transcription factors
Answer b: caspase-8
- 2. The activation of primitive stem cells so as to replace a damaged tissue is called
- a. healing
- b. repair
- c. restoration
- d. regeneration
- e. rejuvenation
Answer d: regeneration
- 3. Defective apoptosis may contribute to the development of
- a. cancer
- b. Alzheimer’s disease
- c. myocardial infarction
- d. aplastic anemia
- e. stroke
Answer a: cancer
- 4. The activation of caspase-3 leads to
- a. activation of transcription factors
- b. inhibition of transcription factors
- c. activation of the anti-apoptotic pathway
- d. fragmentation of genomic DNA
- e. stabilization of cell membranes
Answer d: fragmentation of genomic DNA
- 5. Bortemozib causes the accumulation of an apoptotic-promoter protein by inhibiting
- a. mitochondrial DNA
- b. suvivin
- c. matrix metalloproteinases
- d. pro-apoptotic Bcl-2
- e. the proteasome
Answer e: the proteasome
- 6. Matrix metalloproteinases are responsible for
- a. inhibition of neoplastic cell growth
- b. adjusting the cell cytoskeleton
- c. degradation of extracellular matrix
- d. initiating angiogenesis
- e. activation of the cell cycle
Answer c: degradation of extracellular matrix
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